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Liposomal BPC-157 + KPV

Targeted inflammation and gut-immune modulation with antimicrobial support

Liposomal BPC-157 + KPV combines regenerative repair with intracellular anti-inflammatory and gut-protective signaling for patients with inflammation-driven or microbiome-related concerns.

Key benefits:

  • Support for gastrointestinal & gut barrier integrity* 
  • Anti-microbial support for multiple pathologies*
  • Supports multi-tissue repair, including skin & vascular support*
  • Supports immune & inflammatory-modulating actions*
  • Aids metabolically driven inflammatory dysfunction*
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    • This formulation pairs BPC-157 with KPV, a tripeptide derived from α-MSH, known for its potent anti-inflammatory activity without melanogenic effects.

      What differentiates this formula is KPV’s intracellular mechanism of action:

      • Inhibits NF-κB and MAPK inflammatory pathways inside the cell nucleus
      • Utilizes the PepT1 transporter, enhancing uptake in intestinal epithelial cells
      • Demonstrates antimicrobial activity against common bacterial and fungal strains
      • Supports epithelial barrier repair and immune homeostasis in gut-associated tissue

      Clinically, this product is most appropriate when inflammation is the dominant driver — particularly in gastrointestinal, immune-mediated, or inflammatory skin contexts — and where standard BPC-157 support may be insufficient alone. 

    This formulation pairs BPC-157 with KPV, a tripeptide derived from α-MSH, known for its potent anti-inflammatory activity without melanogenic effects.

    What differentiates this formula is KPV’s intracellular mechanism of action:

    • Inhibits NF-κB and MAPK inflammatory pathways inside the cell nucleus
    • Utilizes the PepT1 transporter, enhancing uptake in intestinal epithelial cells
    • Demonstrates antimicrobial activity against common bacterial and fungal strains
    • Supports epithelial barrier repair and immune homeostasis in gut-associated tissue

    Clinically, this product is most appropriate when inflammation is the dominant driver — particularly in gastrointestinal, immune-mediated, or inflammatory skin contexts — and where standard BPC-157 support may be insufficient alone.